NURS 6501 Week 2 Module 1 Assignment Case Study Analysis

Walden University NURS 6501 Week 2 Module 1 Assignment Case Study Analysis-Step-By-Step Guide

This guide will demonstrate how to complete the Walden University NURS 6501 Week 2 Module 1 Assignment Case Study Analysis assignment based on general principles of academic writing. Here, we will show you the A, B, Cs of completing an academic paper, irrespective of the instructions. After guiding you through what to do, the guide will leave one or two sample essays at the end to highlight the various sections discussed below.

How to Research and Prepare for NURS 6501 Week 2 Module 1 Assignment Case Study Analysis                     

Whether one passes or fails an academic assignment such as the Walden University NURS 6501 Week 2 Module 1 Assignment Case Study Analysis depends on the preparation done beforehand. The first thing to do once you receive an assignment is to quickly skim through the requirements. Once that is done, start going through the instructions one by one to clearly understand what the instructor wants. The most important thing here is to understand the required format—whether it is APA, MLA, Chicago, etc.

After understanding the requirements of the paper, the next phase is to gather relevant materials. The first place to start the research process is the weekly resources. Go through the resources provided in the instructions to determine which ones fit the assignment. After reviewing the provided resources, use the university library to search for additional resources. After gathering sufficient and necessary resources, you are now ready to start drafting your paper.

How to Write the Introduction for NURS 6501 Week 2 Module 1 Assignment Case Study Analysis                     

The introduction for the Walden University NURS 6501 Week 2 Module 1 Assignment Case Study Analysis is where you tell the instructor what your paper will encompass. In three to four statements, highlight the important points that will form the basis of your paper. Here, you can include statistics to show the importance of the topic you will be discussing. At the end of the introduction, write a clear purpose statement outlining what exactly will be contained in the paper. This statement will start with “The purpose of this paper…” and then proceed to outline the various sections of the instructions.

How to Write the Body for NURS 6501 Week 2 Module 1 Assignment Case Study Analysis                     

After the introduction, move into the main part of the NURS 6501 Week 2 Module 1 Assignment Case Study Analysis assignment, which is the body. Given that the paper you will be writing is not experimental, the way you organize the headings and subheadings of your paper is critically important. In some cases, you might have to use more subheadings to properly organize the assignment. The organization will depend on the rubric provided. Carefully examine the rubric, as it will contain all the detailed requirements of the assignment. Sometimes, the rubric will have information that the normal instructions lack.

Another important factor to consider at this point is how to do citations. In-text citations are fundamental as they support the arguments and points you make in the paper. At this point, the resources gathered at the beginning will come in handy. Integrating the ideas of the authors with your own will ensure that you produce a comprehensive paper. Also, follow the given citation format. In most cases, APA 7 is the preferred format for nursing assignments.

How to Write the Conclusion for NURS 6501 Week 2 Module 1 Assignment Case Study Analysis                     

After completing the main sections, write the conclusion of your paper. The conclusion is a summary of the main points you made in your paper. However, you need to rewrite the points and not simply copy and paste them. By restating the points from each subheading, you will provide a nuanced overview of the assignment to the reader.

How to Format the References List for NURS 6501 Week 2 Module 1 Assignment Case Study Analysis                     

The very last part of your paper involves listing the sources used in your paper. These sources should be listed in alphabetical order and double-spaced. Additionally, use a hanging indent for each source that appears in this list. Lastly, only the sources cited within the body of the paper should appear here.

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NURS 6501 Week 2 Module 1 Assignment Case Study Analysis

Sample Answer for NURS 6501 Week 2 Module 1 Assignment Case Study Analysis Included After Question

Case Study Analysis

A 42-year old male comes into the emergency presenting with the following symptoms: a fever of 100.6˚ F, chills and a swollen leg with redness. The patient describes that he was working in his yard when his string trimmer slipped and cut his leg. The patient cleaned the wound with water from his garden hose and covered the wound with a Band-aid. The purpose of this review is to explore why the patient might have the symptoms described, which genes are associated with the development of the disease and provide a description of the immunosuppression process, including its effect on normal body function and systems.

A Sample Answer For the Assignment: NURS 6501 Week 2 Module 1 Assignment Case Study Analysis

Title: NURS 6501 Week 2 Module 1 Assignment Case Study Analysis

NURS 6501 Week 2 Module 1 Assignment Analyze a Case Study

Physiological Modifications

The signs and symptoms are a result of the infection with which the patient was diagnosed, which caused the patient to become septic. Increased blood flow to the area results in the redness seen in the wound. Due to the increased number of neutrophils, white blood cells, nutrient factors, and enzymes deposited in the infected area to aid in the fight against the infection, the area will swell (Hughes, 2016). Chemicals emitted by white blood cells, combined with increased activity at

the site, may result in localized heat and tenderness. Interleukin 10 (IL 10) is a pro-inflammatory cytokine that protects the host and promotes normal tissue homeostasis. Additionally, it is known as Cytokine Synthesis Inhibitory Factor (CSIF).

NRAMP1 is primarily expressed in macrophages and monocytes and is involved in iron metabolism and host resistance to specific pathogens. It also regulates natural resistance to infection. IL 4 is a cytokine that induces young helper T cells to differentiate into Th2 cells (Cianfarani, Zambruno, Castiglia, & Odorisio, 2017).

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Interleukin 4 has anti-inflammatory properties and inhibits tumor necrosis factor production. Immunosuppression can result in an increased susceptibility to opportunistic infections due to an immune deficiency in the body, and even the tiniest pathogens can become pathogenic when the immune system is compromised (Larouche, Sheoran, Maruyama, & Martino, 2018). Immunosuppression may be induced in the body to decrease the risk of allograft rejection and increase the likelihood of survival following transplantation. Allograft vasculopathy may develop spontaneously.

References

Top of the Line

M. A. Hughes (March 24, 2016).
Wound infection: a knowledge gap that must be addressed
British Journal of Nursing, Volume 25, Number 6.)
DOI: 10.12968/bjon.2016.25.6.S46

The case study focuses on a 14 –year –old female who required urgent care. The patient was in the company of her mother. The mother stated that the girl was facing an abnormal number of funny-looking red spotless and had an abnormal number of bruises on her legs. However, the mother objected that the trauma was related to the bruises noted after two weeks. She also noted that the girl had been experiencing a bad rest at home for the past three weeks, led by a bad case of mono. The girl was identified to have bleeding gums when brushing her teeth in the morning. The lab report identified that the girl had a normal WBC differential and hgb and hct.

However, platelet abnormal lab results are based on the platelet count of 100,000/mm3. Bleeding in her gums was also noticed when she brushed her teeth in the morning. A complete workup on the peripheral blood smear immune thrombocytopenic purpura was recommended at the ED. The paper focuses on factors that interfere with fertility and gives the reason for the rise of STD/PID due to the inflammatory markers.

Pathophysiology of Immune Thrombocytopenia Purpose (ITP)

According to Chen et al. (2022), immune thrombocytopenia purpura (ITP) pathophysiology is complex since it consists of T-cell and B-cell abnormalities. There are four causes of thrombocytopenia mechanism, which are consumption, hyperproliferation, destruction, and sequestration. In addition, the mechanism involves a significant proportion of cases and increases platelet destruction, which is an impaired platelet production. Therefore, the megakaryopoiesis and thrombopoiesis defect is experienced due to increased platelet destruction (Grodzielski et al., 2019).

The phagocytosis process helps remove the sanitized platelet, which occurs in the sequestration of the anti-platelet IgG antibodies (Carter, 2018). The sanitized platelet happens a few hours compared to the normal platelet half-life of 8 to 9 days. The drug-induced ITP absorption happens in the platelet cell membrane (McCance & Huether, 2019). Therefore, the autoantibodies react against the platelet glycoproteins. Platelet destruction can also occur when abnormal apoptosis occurs in the bone marrow (McCance & Huether, 2019).

Clinical Manifestation of Immune Thrombocytopenia Purpose (ITP)

Initially, ITP was perceived as a minor injury. However, the purpura and petechiae development occurred every several days, leading to an increase in bleeding from the mucosal site (McCance & Huether, 2019). Patients with ITP show signs like blood in the urine, bleeding gums, and increased bleeding. The main cause of accelerated platelet consumption is increased splenic sequestration or decreased bone marrow production (McCance & Huether, 2019). The lab test helps predict the diagnosis, evaluating the anti-glycoprotein erythrocyte and leukocyte count.

Genetic/Ethnic Considerations of Immune Thrombocytopenia Purpose (ITP)

ITP highly occurs in women (Kjaer et al., 2020). In comparison to acute, the chronic ITP is progressively worse. Acute ITP lasts for approximately one to two months and is common in children (Kjaer et al., 2020). It is not common for a patient to present the intracranial bleeding for the organs or any other sites.ITP is also likely to occur in the newborn as thrombocytopenia.

Conclusion

The patient presents ITP, which is associated with the previous diagnosis of Mononucleosis. It is important to review a patient platelet, which helps determine the anti-platelet antibodies when performing a diagnostic test. ITP highly occurs in women between 20 to 40 years.

References

Carter, C. M. (2018). Alterations in blood components. Comprehensive Toxicology, 249. https://www.ncbi.nlm.nih.gov/pmc/articles/pmc7152208/

Chen, D. P., Lin, W. T., Wen, Y. H., & Wang, W. T. (2022). Investigation of the correlation between immune thrombocytopenia and T cell activity-regulated gene polymorphism using functional study. Scientific Reports, 12(1), 1-8. https://www.nature.com/articles/s41598-022-10631-z

Grodzielski, M., Goette, N. P., Glembotsky, A. C., Constanza Baroni Pietto, M., Méndez-Huergo, S. P., Pierdominici, M. S., … & Marta, R. F. (2019). Multiple concomitant mechanisms contribute to low platelet count in patients with immune thrombocytopenia. Scientific Reports, 9(1), 1-10. https://www.nature.com/articles/s41598-018-38086-1

Kjær, M., Geisen, C., Akkök, Ç. A., Wikman, A., Sachs, U., Bussel, J. B., … & Skogen, B. (2020). Strategies to develop a prophylaxis for the prevention of HPA-1a immunization and fetal and neonatal alloimmune thrombocytopenia. Transfusion and Apheresis Science, 59(1), 102712. https://www.sciencedirect.com/science/article/pii/S147305021930285X

McCance, K. L. & Huether, S. E. (2019). Pathophysiology: The biologic basis for disease in adults and children (8th ed.). St. Louis, MO: Mosby/Elsevier

Top of the Line

F. Cianfarani, G. Zambruno, D. Castiglia, et al (July 01, 2017).
Altered Wound Healing Pathophysiology in Recessive Dystrophic Epidermolysis Bullosa
American Journal of Pathology, vol. 187, no. 7, pp. 1445-1453.
https://doi.org/10.1016/j.ajpath.2017.03.003

Top of the Line

J. Larouche, S. Sheoran, K. Maruyama, and M. M. Martino (July 01, 2018).
Mechanisms and Novel Therapeutic Targets in Immune Regulation of Skin Wound Healing.
Advances in Wound Care, 7, no. 7 (2007), pp. 209-231.
https://doi.org/10.1089/wound.2017.0761

The case study depicts a 42-year-old male who comes to the ED with a two-day history of pain during urination, low back pain, inability to fully empty the bladder, severe perineal pain, fever, and chills. The pain worsens when he stands up and is relieved to some degree by lying down. He has a fever, tachycardia, and tachypnea. The patient has an enlarged, tender, and swollen prostate that is warm to touch on DRE. The purpose of this paper is to discuss Prostatitis as it relates to this patient.

Why Prostatitis and Infection Happens

Prostatitis is an infection of the prostate characterized by painful inflammation of the prostate. Acute bacterial Prostatitis occurs when pathogens enter the prostate gland through the urethra via the prostatic ducts or intraprostatic reflux of urine. Pathogens may also get to the prostate through direct inoculation, for instance, by prostate biopsy or transurethral procedures like cystoscopy and catheterization (Zhang et al., 2020). Prostatitis is mostly caused by Escherichia coli. Other causative organisms include Pseudomonas, Klebsiella, Proteus, and Enterococcus species (Karami et al., 2022). Patients with a lower urinary tract infection (UTI) have a higher risk since pathogens travel from the lower genital tract through the urethra to the prostate, causing inflammation.

The patient has symptoms characteristic of lower UTI, like dysuria, inability to fully empty the bladder, fever, chills, and tachycardia. The pathogens likely traveled to the prostate through the urethra. This caused prostate inflammation, evidenced by DRE findings of an enlarged, tender, and swollen prostate that is warm to touch. Prostatic tenderness indicates bacterial infection (Kanani et al., 2021). The patient presents with clinical manifestations of Acute Bacterial Prostatitis like perineal pain, low back pain, and urinary retention with an inability to void

Tissue invasion occurs in bacterial Prostatitis, which presents with systemic symptoms like fever, chills, malaise, and muscle pain. Furthermore, a generalized sepsis syndrome may occur, presenting with tachypnea, tachycardia, and sometimes hypotension (Kanani et al., 2021). The patient has systemic symptoms like tachypnea, tachycardia, fever, and chills.

Conclusion

Bacterial Prostatitis occurs when pathogens migrate from the lower urinary tract to the prostate through the urethra. The pathogens can also be inoculated during treatment or catheterization. This causes inflammation of the prostate, causing enlargement, tenderness, and swelling. Acute bacterial Prostatitis often causes systemic symptoms of fever, chills, malaise, tachycardia, and tachypnea.

 

 

References

Kanani, S., Mujtaba, N., & Sadler, P. (2021). Acute and chronic prostatitis. InnovAiT, 14(1), 33-37. https://doi.org/10.1177/1755738020966359

Karami, A. A., Javadi, A., Salehi, S., Nasirian, N., Maali, A., Bakhshalizadeh Shadkam, M., Najari, M., Rousta, Z., & Alizadeh, S. A. (2022). Detection of bacterial agents causing prostate infection by culture and molecular methods from biopsy specimens. Iranian journal of microbiology, 14(2), 161–167. https://doi.org/10.18502/ijm.v14i2.9182

Zhang, J., Liang, C., Shang, X., & Li, H. (2020). Chronic prostatitis/chronic pelvic pain syndrome: a disease or symptom? Current perspectives on diagnosis, treatment, and prognosis. American Journal of Men’s Health, 14(1), 1557988320903200. https://doi.org/10.1177/1557988320903